At the university of Rome tor Vergata. The aim of the Lab is to define molecular mechanisms governing programmed cell death. And to reveal program alterarions that can lead to disease. We are currently focusing on a monogenic disease - Friedreich’s Ataxia. In which the deficiency of the mitochondrial protein frataxin. Causes enhanced susceptibility to stress-induced cell death. Directed by prof. roberto testi. Follow us on FB.
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Mission | cellwideweb.com Reviews
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At the university of Rome tor Vergata. The aim of the Lab is to define molecular mechanisms governing programmed cell death. And to reveal program alterarions that can lead to disease. We are currently focusing on a monogenic disease - Friedreich’s Ataxia. In which the deficiency of the mitochondrial protein frataxin. Causes enhanced susceptibility to stress-induced cell death. Directed by prof. roberto testi. Follow us on FB.
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Friedreich's Ataxia, a degenerative disease due to accelerated cell death. We selected a human monogenic disease in which accelerated cell death in specific tissues has pathogenic relevance. Friedreich's Ataxia (FRDA) is caused by the defective expression
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Friedreich's Ataxia, a degenerative disease due to accelerated cell death. We selected a human monogenic disease in which accelerated cell death in specific tissues has pathogenic relevance. Friedreich's Ataxia. FRDA) is caused by the defective expression of frataxin. A mitochondrial protein involved in iron homeostasis. We defined the precise size of mature frataxin. A major player in iron metabolism. More recently, we identified the E3 ligase that directly ubiquitinates the frataxin precursor, RNF126.
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E3 Ligase RNF126 Directly Ubiquitinates Frataxin, Promoting Its Degradation: Identification of a Potential Therapeutic Target for Friedreich Ataxia. Benini M, Fortuni S, Condò I, Alfedi G, Malisan F, Toschi N, Serio D, Massaro DS, Arcuri G, Testi R, Rufini A. Cell Rep. 2017 Feb 21;18(8):2007-2017. Analyzing the Effects of a G137V Mutation in the FXN Gene. Faggianelli N, Puglisi R, Veneziano L, Romano S, Frontali M, Vannocci T, Fortuni S, Testi R, Pastore A. Front Mol Neurosci. 2015 Nov 25;8:66. Autophagy...
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The Laboratory of Signal Transduction has been recently funded by:.
VI FP - Life Sciences, 2004-2007Programmed cell death across the eukaryotic kingdoms (TRANSDEATH) The ObjectivesThe TransDeath project will focus on cellular and molecular events in less known cell death types. Thus, the project aims to broaden the experi
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VI FP - Life Sciences, 2004-2007. Programmed cell death across the eukaryotic kingdoms (TRANSDEATH). V FP - Quality of Life, 2001-2004. Molecular mechanisms of senescence and ageing (CELLAGE). The goal of this project is to define determinants of healthy ageing and use this knowledge to understand age-related disfunctions, with the ultimate goal to develop new strategies for their prevention and treatment. IV FP - Training and Mobility of Researchers, 1998-2001. IV FP - Biomed 2, 1996-1999. 1) To examine...
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Mission
At the university of Rome tor Vergata. The aim of the Lab is to define molecular mechanisms governing programmed cell death. And to reveal program alterarions that can lead to disease. We are currently focusing on a monogenic disease - Friedreich’s Ataxia. In which the deficiency of the mitochondrial protein frataxin. Causes enhanced susceptibility to stress-induced cell death. Directed by prof. roberto testi. Follow us on FB.
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