At the university of Rome tor Vergata. The aim of the Lab is to define molecular mechanisms governing programmed cell death. And to reveal program alterarions that can lead to disease. We are currently focusing on a monogenic disease - Friedreich’s Ataxia. In which the deficiency of the mitochondrial protein frataxin. Causes enhanced susceptibility to stress-induced cell death. Directed by prof. roberto testi. Follow us on FB.
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At the university of Rome tor Vergata. The aim of the Lab is to define molecular mechanisms governing programmed cell death. And to reveal program alterarions that can lead to disease. We are currently focusing on a monogenic disease - Friedreich’s Ataxia. In which the deficiency of the mitochondrial protein frataxin. Causes enhanced susceptibility to stress-induced cell death. Directed by prof. roberto testi. Follow us on FB.
robertotesti.com
Laboratory of signal transduction
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Rossana Trotta, B.S. Ph.D., Jefferson University, Philadelphia (PA). Daniele D'Ambrosio, M.D. Ph.D., HSR-DIBIT, Roche Ricerche, Milano. Ruggero De Maria, M.D., Istituto Superiore di Sanita', Rome. Ann Zeuner, Ph.D., Istituto Superiore di Sanita', Rome. Maria Rita Mita Rippo, B.S. Ph.D., Universita' di Ancona. Chiara Nicolo', B.S. Ph.D., Universita' Cattolica, Roma. Luigi Franchi, M.D., Ph.D., University of Michigan, Ann Arbor (MI). Valeria Zacchei, Business Manager, BigBerry, Rome. Stefano Fais, ISS, Rome.
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Friedreich's Ataxia, a degenerative disease due to accelerated cell death. We selected a human monogenic disease in which accelerated cell death in specific tissues has pathogenic relevance. Friedreich's Ataxia (FRDA) is caused by the defective expression
http://www.robertotesti.com/Site/Topics.html
Friedreich's Ataxia, a degenerative disease due to accelerated cell death. We selected a human monogenic disease in which accelerated cell death in specific tissues has pathogenic relevance. Friedreich's Ataxia. FRDA) is caused by the defective expression of frataxin. A mitochondrial protein involved in iron homeostasis. We defined the precise size of mature frataxin. A major player in iron metabolism. More recently, we identified the E3 ligase that directly ubiquitinates the frataxin precursor, RNF126.
VI FP - Life Sciences, 2004-2007Programmed cell death across the eukaryotic kingdoms (TRANSDEATH) The ObjectivesThe TransDeath project will focus on cellular and molecular events in less known cell death types. Thus, the project aims to broaden the experi
http://www.robertotesti.com/Site/EU_Projects.html
VI FP - Life Sciences, 2004-2007. Programmed cell death across the eukaryotic kingdoms (TRANSDEATH). V FP - Quality of Life, 2001-2004. Molecular mechanisms of senescence and ageing (CELLAGE). The goal of this project is to define determinants of healthy ageing and use this knowledge to understand age-related disfunctions, with the ultimate goal to develop new strategies for their prevention and treatment. IV FP - Training and Mobility of Researchers, 1998-2001. IV FP - Biomed 2, 1996-1999. 1) To examine...
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The Laboratory of Signal Transduction has been recently funded by:.
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At the university of Rome tor Vergata. The aim of the Lab is to define molecular mechanisms governing programmed cell death. And to reveal program alterarions that can lead to disease. We are currently focusing on a monogenic disease - Friedreich’s Ataxia. In which the deficiency of the mitochondrial protein frataxin. Causes enhanced susceptibility to stress-induced cell death. Directed by prof. roberto testi. Follow us on FB.
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Just another WordPress.com weblog. Los primeros años de un nuevo orden global ( y México? Bull;mayo 20, 2009 • Dejar un comentario. El contenido nos muestra los cambios sufridos por cuatro países: Estados Unidos de América (USA), la Unión de Repúblicas Socialistas Soviéticas (URSS), la República Popular de China (China) y la República de India (India). Pero es en los años 90, cuando abruptamente surge la necesidad de concretar un nuevo orden mundial, para el que las personas y las organizaciones sociales...
