spandidos-publications.org
Spandidos PublicationsSpandidos Publications is a scientific publisher with a long-standing international reputation for excellent standards and high quality science publications
http://www.spandidos-publications.org/
Spandidos Publications is a scientific publisher with a long-standing international reputation for excellent standards and high quality science publications
http://www.spandidos-publications.org/
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Spandidos Publications
Dimitrios Spandidos
10, Vri●●●●●●●Street,
At●●ns , Athens, 116-35
GR
View this contact
Spandidos Publications
Dimitrios Spandidos
10, Vri●●●●●●●Street,
At●●ns , Athens, 116-35
GR
View this contact
Spandidos Publications
Dimitrios Spandidos
10, Vri●●●●●●●Street,
At●●ns , Athens, 116-35
GR
View this contact
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https://spandidos-publications.org
Spandidos Publications is a scientific publisher with a long-standing international reputation for excellent standards and high quality science publications
spandidos-publications.org
Spandidos Publications
http://www.spandidos-publications.org/pages/abstracting
Biomedical Report is indexed and/or abstracted in:. Emerging Sources Citation Index (ESCI, Thomson ISI). Chemical Abstracts Service (CAS). OCLC (WorldCat Local and WorldCat.org).
Spandidos Publications
http://www.spandidos-publications.org/pages/editorialPol
1 Publication and authorship. 2 Peer review/responsibility for the reviewers. 1 Publication and authorship. Plagiarism and other fraud. If a case of plagiarism comes to light following publication of a manuscript, the journal will conduct a preliminary investigation. If plagiarism is detected, the manuscript containing the plagiarism will be obviously marked on each page of the PDF. Depending on the extent of the plagiarism, the paper may also be formally retracted. Ethical conduct of research. Authors a...
Spandidos Publications
http://www.spandidos-publications.org/pages/info_for_librarians
We offer print and online subscriptions to the International Journal of Molecular Medicine, International Journal of Oncology, Molecular Medicine Reports, Oncology Reports, Experimental and Therapeutic Medicine, Oncology Letters, Biomedical Reports and Molecular and Clinical Oncology. INT J MOL MED. For Information regarding each journal, please download the " 2017 Library Catalogue.". 1 Institutional subscriptions›. Total FTE = total number of staff and students across all sites. Spandidos Publications ...
Spandidos Publications
http://www.spandidos-publications.org/pages/info_for_authors
The content of this webpage is available for download as a PDF. The content of this webpage is available for download in PDF. Spandidos Publications journals utilize an online submission and tracking system designed to provide a faster, more efficient service to authors. The principal aim of Spandidos Publications is to publish promptly original works of high quality in English. Spandidos Publications utilizes iThenticate to screen submitted manuscripts against published studies and other relevant source...
Spandidos Publications
http://www.spandidos-publications.org/pages/conference
21st World Congress on Advances in Oncology. 19th International Symposium on Molecular Medicine. October 6-8, 2016. Metropolitan Hotel, Athens, Greece. Digital PCR - Applications and analysis. October 4-5, 2016. Metropolitan Hotel, Athens, Greece. Registration and Accommodation Information. Pre-Congress Workshop 2016 (Digital PCR). 2nd Workshop on Paediatric Virology 2016 (October 8th). Conference Sights and Maps of Athens. Metropolitan Hotel (5* Conference Center). Coral Hotel Athens (4*).
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Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
Spandidos Publications
Volume 36 Issue 3. Print ISSN : 1107-3756. 2014 Impact Factor: 2.088. Ranked #22/123 Medicine Research and Experimental. Total number of cites). Asparaginase treatment side-effects may be due to genes with homopolymeric Asn codons (Review-Hypothesis). MiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling. Autophagy attenuates the catabolic effect during inflammatory conditions in nucleus pulposus cells, as sustained by NF-κB and JNK inhibition. Volume 47 Issue 3.
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