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Tuberous Sclerosis

TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析TUBERIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示TUBERIN的Ser939、Ser1130與Thr1462很可能會受AKT磷酸化。其中Ser939與Thr1462已被證實會受AKT磷酸化，且TUBERIN的活性將因此而被抑制。 TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析HAMARTIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示HAMARTIN蛋白質無明顯會被AKT磷酸化的可能位置，由此可推測AKT主要調控的對象可能是TUBERIN蛋白質。 TUBERIN-Pattern and profile search. 藉由MotifScan ( http:/ myhits.isb-sib.ch/cgi-bin/motif scan. HAMARTIN-Pattern and profile search.

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Tuberous Sclerosis | tuberoussclerosis.blogspot.com Reviews

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TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析TUBERIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示TUBERIN的Ser939、Ser1130與Thr1462很可能會受AKT磷酸化。其中Ser939與Thr1462已被證實會受AKT磷酸化，且TUBERIN的活性將因此而被抑制。 TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析HAMARTIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示HAMARTIN蛋白質無明顯會被AKT磷酸化的可能位置，由此可推測AKT主要調控的對象可能是TUBERIN蛋白質。 TUBERIN-Pattern and profile search. 藉由MotifScan ( http:/ myhits.isb-sib.ch/cgi-bin/motif scan. HAMARTIN-Pattern and profile search.

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Tuberous Sclerosis | tuberoussclerosis.blogspot.com Reviews

https://tuberoussclerosis.blogspot.com

TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析TUBERIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示TUBERIN的Ser939、Ser1130與Thr1462很可能會受AKT磷酸化。其中Ser939與Thr1462已被證實會受AKT磷酸化，且TUBERIN的活性將因此而被抑制。 TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析HAMARTIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示HAMARTIN蛋白質無明顯會被AKT磷酸化的可能位置，由此可推測AKT主要調控的對象可能是TUBERIN蛋白質。 TUBERIN-Pattern and profile search. 藉由MotifScan ( http:/ myhits.isb-sib.ch/cgi-bin/motif scan. HAMARTIN-Pattern and profile search.

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tuberoussclerosis.blogspot.com

1

Tuberous Sclerosis: HAMARTIN-Family information

http://tuberoussclerosis.blogspot.com/2009/01/hamartin-family-information.html

基因經轉譯作用 (translation) 產生的蛋白質。搜尋Inter-. ProScan Sequence Search ( http:/ www.ebi.ac.uk/Tools/InterProScan/. 資料庫，得知hamartin在細胞中參與調控的兩條訊息傳遞路徑：1. p53 path-. Way by glucose deprivation。2. Insulin/IGF pathway-protein kinase B sign-. 訂閱：張貼留言 (Atom). Introduction of tuberous sclerosis (MIM 191100). Molecular mechanism of tuberous sclerosis. TSC1 gene (MIM 605284). TSC2 gene (MIM 191092). TSC1 and TSC2 proteins alignment. P53 pathway by glucose deprivation. Insulin/IGF pathway-protein kinase B signaling cas.

2

Tuberous Sclerosis: Molecular mechanism of tuberous sclerosis

http://tuberoussclerosis.blogspot.com/2009/01/molecular-mechanism-of-tuberous.html

Molecular mechanism of tuberous sclerosis. 兩個抑癌基因 (tumor suppressor gene) 的突變所造成。兩基因生成的蛋白質會結合形成復合體，進而影響下游蛋白質的活性。如圖所示，. 基因的蛋白質複合體會調控mTOR (mammalian target of rapamycin) 訊息傳遞路徑，進而調控細胞增生。若. 圖片來源: http:/ www.biocarta.com/pathfiles/h mtorPathway.asp. 訂閱：張貼留言 (Atom). Introduction of tuberous sclerosis (MIM 191100). Molecular mechanism of tuberous sclerosis. TSC1 gene (MIM 605284). TSC2 gene (MIM 191092). TSC1 and TSC2 proteins alignment. P53 pathway by glucose deprivation. HAMARTIN-Pattern and profile search.

3

Tuberous Sclerosis: TUBERIN-Phosphorylation site prediction

http://tuberoussclerosis.blogspot.com/2009/01/tuberin-phosphorylation-site-prediction.html

TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析TUBERIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示TUBERIN的Ser939、Ser1130與Thr1462很可能會受AKT磷酸化。其中Ser939與Thr1462已被證實會受AKT磷酸化，且TUBERIN的活性將因此而被抑制。訂閱：張貼留言 (Atom). Introduction of tuberous sclerosis (MIM 191100). Molecular mechanism of tuberous sclerosis. TSC1 gene (MIM 605284). TSC2 gene (MIM 191092). TSC1 and TSC2 proteins alignment. P53 pathway by glucose deprivation. Insulin/IGF pathway-protein kinase B signaling cas.

4

Tuberous Sclerosis: HAMARTIN-Leucine zipper prediction

http://tuberoussclerosis.blogspot.com/2009/01/hamartin-leucine-zipper-prediction.html

利用2ZIP ( http:/ 2zip.molgen.mpg.de/index.html. 分析HAMARTIN的胺基酸序列，搭配COILS ( http:/ www.ch.embnet.org/software/COILS form.html. 的分析結果，推測HAMARTIN接近C端 (C-terminus) 的區域可能具有包含白胺酸 (leucine) 的α螺旋 (α-helix)，但不足以形成白胺酸拉鏈 (leucine zipper) 的模式 (motif)。推測此結構乃是用於與TUBERIN上包含白胺酸的α螺旋結合，形成白胺酸拉鏈的模式，藉此使HAMARTIN與TUBERIN形成蛋白質複合體 (protein complex)。訂閱：張貼留言 (Atom). Introduction of tuberous sclerosis (MIM 191100). Molecular mechanism of tuberous sclerosis. TSC1 gene (MIM 605284). TSC2 gene (MIM 191092).

5

Tuberous Sclerosis: Insulin/IGF pathway-protein kinase B signaling cascade

http://tuberoussclerosis.blogspot.com/2009/01/insulinigf-pathway-protein-kinase-b.html

Insulin/IGF pathway-protein kinase B signaling cascade. 當細胞接受胰島素 (insulin) 或類胰島素生長因子 (insulin-like growth factor；IGF)，AKT將抑制TSC1/2複合體 (complex) 的活性，使細胞增生不受抑制。雖然AMPK與AKT皆可將TSC1/2複合體磷酸化，但根據作用的位置不同，可活化或抑制TSC1/2複合體的活性。圖片來源: http:/ www.ebi.ac.uk/Tools/InterProScan/. 訂閱：張貼留言 (Atom). Introduction of tuberous sclerosis (MIM 191100). Molecular mechanism of tuberous sclerosis. TSC1 gene (MIM 605284). TSC2 gene (MIM 191092). TSC1 and TSC2 proteins alignment. P53 pathway by glucose deprivation.

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tuberoussclerosis.blogspot.com

Tuberous Sclerosis

TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析TUBERIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示TUBERIN的Ser939、Ser1130與Thr1462很可能會受AKT磷酸化。其中Ser939與Thr1462已被證實會受AKT磷酸化，且TUBERIN的活性將因此而被抑制。 TUBERIN蛋白質複合體 (protein complex) 的活性。在此以Group-based Prediction System (GPS 2.1) 分析HAMARTIN的胺基酸序列中，可能被AKT磷酸化的位置。結果顯示HAMARTIN蛋白質無明顯會被AKT磷酸化的可能位置，由此可推測AKT主要調控的對象可能是TUBERIN蛋白質。 TUBERIN-Pattern and profile search. 藉由MotifScan ( http:/ myhits.isb-sib.ch/cgi-bin/motif scan. HAMARTIN-Pattern and profile search.

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